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Your search term(s) "celiac disease" returned 309 results.

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Gluten-Sensitive Enteropathy (Celiac Disease): More Common Than You Think. American Family Physician. 66(12): 2259-2266. December 15, 2002.

This article describes gluten-sensitive enteropathy or, as it is more commonly called, celiac disease, an autoimmune inflammatory disease of the small intestine that is precipitated by the ingestion of gluten, a component of wheat protein, in genetically susceptible persons. Exclusion of dietary gluten results in healing of the mucosa, resolution of the malabsorptive state, and reversal of most, if not all, effects of celiac disease. Recent studies in the United States suggest that the prevalence of celiac disease is approximately one case per 250 percents. Gluten-sensitive enteropathy commonly manifests as 'silent' celiac disease (i.e., minimal or no symptoms). Serologic tests for antibodies against endomysium, transglutaminase, and gliadin identify most patients with the disease. The author stresses that serologic testing should be considered in patients who are at increased genetic risk for gluten sensitive enteropathy (i.e., family history of celiac disease or personal history of type I diabetes) and in patients who have chronic diarrhea, unexplained anemia, chronic fatigue, or unexplained weight loss. Early diagnosis and management are important to forestall serious consequences of malabsorption, such as osteoporosis and anemia. 5 figures. 6 tables. 37 references.

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Guia de Dieta Para Enfermedad Celiac. [Dieting Guide for Celiac Disease]. Studio City, CA: Celiac Disease Foundation. 2002. 4 p.

This Spanish language brochure helps readers recently diagnosed with celiac disease (gluten intolerance) understand the basics of the gluten-free diet used to treat celiac. The brochure reviews the foods that have gluten in them, and lists common foods that are and are not permitted on a gluten-free diet. Readers are cautioned that some less obvious ingredients may have gluten in them, such as caramel coloring, dehydrated vegetable protein, malt vinegar, and salsa. Readers are encouraged to read labels carefully to assist in their efforts to stay gluten-free. One section considers the psychosocial impact of making a major lifestyle change, such as undertaking a gluten-free diet. The brochure concludes with information about the activities and goals of the Celiac Disease Foundation (www.celiac.org).

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Guide to Gluten-Free Beginnings. In: Sanderson, S.L. Incredible Edible Gluten-Free Food for Kids: 150 Family-Tested Recipes. Bethesda, MD: Woodbine House. 2002. p. 1-9.

This introductory chapter, from a cookbook for families coping with celiac disease (gluten intolerance), familiarizes parents with some of the issues they need to address when following a gluten-free diet. The author stresses that the key to the gluten-free diet is to identify and then avoid foods that contain gluten, the insoluble protein component of wheat, rye, barley, and other grains. Since 'gluten' is rarely listed as an ingredient in products that contain gluten, this chapter covers the basics parents need to know to identify gluten-containing foods and keep them from entering the child's diet. The chapter lists allowed and not allowed foods, questionable foods, the importance of continuing one's education about gluten and the gluten-free diet, how to organize the kitchen to support a gluten-free approach, food preparation, and how to reduce stress in food preparation.

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Incredible Edible Gluten-Free Food for Kids: 150 Family-tested Recipes. Bethesda, MD: Woodbine House. 2002. 333 p.

Kids who must follow a gluten-free diet due to celiac disease or another condition need not only food made without wheat, rye, oats, and barley, but tasty food, to help them stick to their diet. This cookbook offers gluten-free recipes formulated especially for children and families. With this cookbook, parents can cook food without gluten that children will actually eat. The author provides an overview of celiac disease, advice about managing meals eaten away from home, entertaining and party tips, trouble-shooting ideas for bakers, metric and temperature conversion tables, and a resource section listing gluten-free suppliers and manufacturers and support groups. Recipes are provided in seven categories: appetizers and snacks, breads, breakfast ideas, lunch and dinner ideas, salads and vegetables, soups and stews, and sweet treats. The cookbook concludes with five indices: all recipes, easy recipes, quick recipes, finger food recipes, and a subject index.

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Longitudinal Study of the Effects of a Gluten-Free Diet on Glycemic Control and Weight Gain in Subjects with Type 1 Diabetes and Celiac Disease. Diabetes Care. 25(7): 1117-1122. July 2002.

This article reports on a study undertaken to describe the longitudinal growth characteristics and glycemic control in children with type 1 diabetes diagnosed with celiac disease and started on a gluten-free diet (GFD). Data on growth and glycemic control for 11 case subjects diagnosed with celiac disease (cd positive group) and started on a GFD were collected prospectively, and two control subjects without celiac disease matched for age, sex, and duration of diabetes (cd negative group) were selected for comparison. In children with type 1 diabetes, untreated celiac disease resulted in lower BMI (body mass index) and lower HbA1c (glycosylated hemoglobin, a measure of blood glucose levels over time). Recovery of BMI with a GFD was associated with further improvement in HbA1c, as compared with pre-GFD, with no expected deterioration in glycemic control during puberty. The authors conclude that these apparent clinical benefits need to be confirmed by larger studies. 2 figures. 2 tables. 33 references.

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My Diabetes Health Record for Kids and Teens. Minneapolis, MN: International Diabetes Center. 2002. (chart).

This fold-out chart, printed on heavy cardstock, offers a pocket-sized way to keep track of one's diabetes health information in one place. The chart, designed for adolescents and children, includes three separate sections. The first section covers general diabetes care, which has space for recording glycosylated hemoglobin (HbA1c), treatment plan review, visual foot exam, comprehensive foot exam, urinary protein, thyroid function, gliadin antibodies for celiac disease, formal diabetes education, retinal eye exam, and dental exam. The second section covers 'heart checkpoints,' including blood pressure, total cholesterol, low density lipoprotein (LDL), high density lipoprotein (HDL), and triglycerides. The third section covers vaccinations. The chart also includes a place to record one's blood type. For each assessment, there are 12 slots labeled with the months of the year, to record the dates the tests were done and the results found.

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Rheumatic Manifestations of Gastrointestinal Diseases. Bulletin on the Rheumatic Diseases. 51(2): 1-3. 2002.

Pathologic changes in the gastrointestinal tract may be associated with clinical complaints in multiple organs, including the musculoskeletal system. This article focuses primarily on the rheumatic manifestations of the gastrointestinal diseases and the available treatment modalities for these problems. The authors note that impairment of the gastrointestinal barrier function may play a role in the pathogenesis (development) of arthropathies. Arthritis is the most common extracolonic (outside the large intestine) manifestation of chronic ulcerative colitis (a type of inflammatory bowel disease). Reactive arthritis is one of the most common arthritides affecting young adults. In some instances, the association between gastrointestinal pathology and extraintestinal disease is so strong that treatment of gastrointestinal disease resolves many of the patient's extraintestinal complaints. The authors discuss inflammatory bowel disease (IBD, including ulcerative colitis and Crohn's idsease), celiac disease (gluten enteropathy), Whipple's disease, and enteric reactive arthritis. The authors conclude that it is important to keep enteropathic arthritis (joint manifestations that occur in conjunction with gastrointestinal disease) in the differential diagnosis of patients with unexplained arthritis and to obtain detailed history, complete physical examination, and appropriate testing before treatment. 2 tables. 37 references.

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Role of Hemochromatosis Susceptibility Gene Mutations in Protecting Against Iron Deficiency in Celiac Disease. Gastroenterology. 123(2): 444-449. August 2002.

Celiac disease and hereditary hemochromatosis (HH) are common HLA-defined conditions in northwestern Europe. This article reports on a study undertaken to determine whether there is a genetic relationship between the 2 diseases and if hemochromatosis susceptibility gene (HFE) mutations are protective against iron deficiency in celiac disease. HFE gene mutations (H63D or C282Y) were identified in 70 celiac patients (48.3 percent) and 61 controls (32.5 percent). The authors conclude that in celiac disease, HFE gene mutations are common and are in linkage disequilibrium with different HLA alleles compared with controls. The authors propose that HFE gene mutations provide a survival advantage by ameliorating the iron deficiency seen in celiac patients. 4 tables. 30 references.

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Sleisenger and Fordtran's Gastrointestinal and Liver Disease: Pathophysiology/Diagnosis/Management. 7th ed. St. Louis, MO: Saunders. 2002. (CD-ROM).

This CD-ROM is an interactive version of a definitive compendium of current clinical knowledge in gastroenterology and hepatology. The authors offer a balanced, detailed account of the basic science of the digestive system, as well as complete coverage of current diagnosis and management. Readers will find up-to-date discussions of the cell biology and molecular biology that determine the digestive system's function, in addition to descriptions of organ physiology and the pathophysiology of the signs, symptoms and laboratory abnormalities of organ disease. The program contains an expanded section on the liver, integrates the latest endoscopic scanning and therapeutic techniques, discusses the latest perspective in possible roles of H.pylori in dyspepsia and gastric cancer, contains information on possible causes of non-ulcer dyspepsia, presents expanded coverage of GI bleeding, with description of the latest techniques for diagnosis and treatment, contains new information on genetic markers for cancer of the colon, post liver transplantation for hepatitis and diagnostic tests for celiac disease, offers a complete description of effective responses for inflammatory bowel disease and Crohn's disease, and discusses relevant histological criteria for the diagnosis of Barrett's esophagus, gastritis, IBD and celiac disease. The program features a separate section on problems involving multiple organs such as AIDS, systemic manifestation of GI disease and abdominal pain and bleeding. The program is illustrated with full-color photographs and drawings.

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Small Intestine and Nutrition: Intestinal Permeation and Gastrointestinal Disease. Journal of Clinical Gastroenterology. 34(4): 385-396. April 2002.

The gastrointestinal tract constitutes one of the largest sites of exposure to the outside environment. Maintenance of host integrity (the health of the body) requires effective monitoring of this site and sealing of the host interior against potential intruders such as bacteria, toxins, or other antigenic molecules; this function is called the gut barrier. This review article introduces some of the basic mechanisms of gut barrier function and the tools used in its assessment. Disruptions in the gut barrier follow injury from various causes including nonsteroidal anti-inflammatory drugs (NSAIDs) and oxidant stress, and involve mechanisms such as adenosine triphosphate depletion and damage to epithelial (lining) cell cystoskeletons that regulate tight junctions. Ample evidence links gut barrier dysfunction to multiorgan system failure in sepsis and immune dysregulation. The authors provide an overview of the evidence for the role of gut barrier dysfunction in disorders such as Crohn's disease, celiac disease, food allergy, acute pancreatitis, non-alcoholic fatty liver disease, and alcoholic liver disease. The authors offer critical insight into the implications of this evidence as a primary disease mechanism. 1 figure. 106 references.

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