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| Benjamin Feldman, Ph.D., Investigator |
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Dr. Benjamin Feldman received his BA in Microbiology and Immunology from UC Berkeley in 1985. He earned his PhD in 1995, in Dr. Mitchell Goldfarb's laboratory at Columbia University, where he studied the genetic requirements for Fibroblast Growth Factor Four in mice. For his postdoctoral studies he worked on Nodal signaling in zebrafish, first in Dr. William Talbot's laboratory at the New York University Medical Center, then continuing in Dr. Derek Stemple's laboratory at the National Institute for Medical Research, London, UK. Dr. Feldman joined the NIH as an Investigator in 2003, where he continues to work with zebrafish to investigate mechanisms of vertebrate gastrulation and to model human diseases.
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Selected Recent Publications:
Feldman B, Concha M, Saúde L, Parsons MJ, Adams R, Wilson S, and Stemple DL. (2002) Lefty antagonism of squint is essential for normal gastrulation, Current Biology 12, 2129-2135.
Full Text/Abstract
Parsons MJ, Pollard SM, Saude L, Feldman B, Coutinho P, Hirst EM and (2002) Zebrafish mutants identify an essential role for laminins in notochord formation, Development, 129, 3137-46.
Full Text/Abstract
Feldman B and Stemple DL. (2001) Morpholino phenocopies of sqt, oep and ntl mutations, Genesis 30, 175-177.
Full Text/Abstract
B. Feldman (2001) Meeting review: The Second European Conference on Zebrafish Genetics and Development: University College London, England: April 19-22, 2001., Comparative and Functional Genomics 2, 252-256.
All Selected Publications
Contact Information:
Dr. Benjamin Feldman
Vertebrate Embryology Section, NHGRI
Porter Neuroscience Research Center
Building 35, Room 1B-205
35 Convent Dr, MSC 3717
Bethesda, MD 20892-3717
Telephone: (301) 594-7487 (office),
(301) 435-2040 (laboratory),
(301) 496-7184 (fax)
Email: bfeldman@mail.nih.gov
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