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Either mithocondrial damage or Fas activation mediate apoptosis in HIV-gp120-treated CD4+ T cells.

Piccolella E, di Somma MM, Somma F, Gilardini Montani MS; International Conference on AIDS.

Int Conf AIDS. 1998; 12: 272 (abstract no. 21166).

BACKGROUND: We have recently published that the susceptibility to gp120-mediated apoptosis of CD4+ T lymphocytes repeatedly antigen stimulated is regulated by bcl-2 expression, cell cycle progression and costimulatory signals but not by Fas expression. However, Fas pathway activation has been described as responsible, at least in part, of apoptotic phenomena in HIV-infected individuals. All together these results drove us to hypothesize that both Fas-dependent and -independent apoptotic pathways could mediate apoptosis in AIDS. METHODS: We tested this hypothesis by using CD4+ T cell clones, induced to apoptosize by antigen specific activation following pretreatment with soluble gp120. Fas and fasL mRNA expression by RT-PCR, Fas-mediated apoptosis by agonistic antibodies or soluble recombinant FasL or FasL+ bystander cells, caspase activity by western blot and mitochondrial membrane potential by fluorescent probe JC-1 were analyzed in these cells. Moreover the protective role of IL-2 and IL-4 was also investigated in the same system. RESULTS: We demonstrated that: a) the interaction of gp120 with CD4 inhibits FasL expression and favors Fas-mediated activation signals, b) gp120 mediates apoptotic programs by a caspase independent way, unless Fas molecules are triggered by specific ligands, and c) mitochondrial damage is involved in Fas-independent apoptotic pathway. CONCLUSION: Our data indicate that gp120 programs CD4+ T cells to apoptosis both via a caspase independent mechanism, inhibited by IL-2 and IL-4 mediated bcl-2 increase, and via a caspase dependent mechanism triggered by FasL+ bystander cells.

Publication Types:
  • Meeting Abstracts
Keywords:
  • AIDS Vaccines
  • Acquired Immunodeficiency Syndrome
  • Antigens, CD4
  • Apoptosis
  • CASP4 protein, human
  • CD4-Positive T-Lymphocytes
  • Caspases
  • Fas Ligand Protein
  • HIV Envelope Protein gp120
  • HIV Infections
  • HIV Seropositivity
  • Interleukin-2
  • Interleukin-4
  • Membrane Glycoproteins
  • T-Lymphocytes
  • immunology
Other ID:
  • 98393897
UI: 102228704

From Meeting Abstracts




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